Hot Products

Ganolactone B
Catalog No: CFN90748

Ganolactone B is a natural product from Ganoderma lucidum.

Ganolucidic acid A
Catalog No: CFN90299

Ganolucidic acid A exhibits cytotoxicity, has inhibitory activities against human HeLa cervical cancer cell lines. It shows significant anti-human immunodeficiency virus (anti-HIV)-1 protease activity with IC50 values of 20-90 microM.

Ganolucidic acid B
Catalog No: CFN95513

Ganolucidic acids B showd induction ability of hPXR-mediated CYP3A4 expression.

Ganolucidic acid E
Catalog No: CFN95536

Ganolucidic acid E inhibits the growth of three types of human cancer cells: Caco-2, HepG2 and HeLa cells.

Ganomycin I
Catalog No: CFN92595

Ganomycin I inhibits the activity of HIV-1 protease.

Garbanzol
Catalog No: CFN96497

Garbanzol is a natural product from Cicer arietinum.

Garcinone C
Catalog No: CFN97254

Garcinone C is a potent inhibitor of AChE. It is an active compound against both of pathogenic (MIC =100 μg/ml) and non-pathogenic leptospira (MIC = 200 μg/ml). Garcinone C exhibits either significant or moderate cytotoxicity against MCF-7, A549, Hep-G2 and CNEhuman cancer cell lines in vitro.

Garcinone D
Catalog No: CFN90383

Garcinone D shows significant cytotoxicity against the CEM-SS cell line, with IC(50) value of 3.2 microg/ml; it exhibits dose-dependent enzyme-based microsomal aromatase inhibitory activity. Garcinone D inhibits p65 activation with IC50 values of 3.2 microM.

Garcinone E
Catalog No: CFN92697

Garcinone E is active constituents in the anticomplement assay used. Garcinone E exhibits potent activity in vitro against Plasmodium falciparum chloroquine-resistant strain W2, with IC50 values below 3 µM. It has potent cytotoxic effect on all hepatocellular carcinomas cell lines as well as on the other gastric and lung cancer cell lines included in the screen, may be potentially useful for the treatment of certain types of cancer.

Gardenin A
Catalog No: CFN70471

Gardenin A has antihyperlipidemic and hepatoprotective effects , it also promotes neuritogenesis through the activation of MAPK/ERK-, PKC-, and PKA-dependent, but not TrkA-dependent, CREB signaling pathways in PC12 cells.